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Updated: Dec 10 2021

Myocardial Infarction

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  • Snapshot
    • A 55-year-old man presents to the emergency department due to substernal chest pain. His symptoms began a few hours ago. He describes the pain as "crushing" and it radiates down the left arm. Medical history is significant for type 2 diabetes and hypertension. On physical exam the patient is diaphoretic. An electrocardiogram demonstrates ST-segment elevations and cardiac troponins are significantly elevated.
  • Introduction
    • Clinical definition
      • death of myocardial tissue secondary to prolonged and severe ischemia
        • also known as a "heart attack"
    • Types
      • ST-segment elevation myocardial infarction (STEMI)
        • an acute coronary syndrome (ACS) with ST-segment elevations found on electrocardiogram (ECG)
        • biomarkers of myocardial necrosis are present
      • Non-STEMI (NSTEMI)
        • an ACS without ST-segment elevations found on ECG
        • biomarkers of myocardial necrosis are present
    • Epidemiology
      • incidence
        • increases with age
      • risk factors
        • hypertension
        • cigarette smoking
        • hyperlipidemia
        • hypercholesterolemia
        • male
        • postmenopause
    • Etiology
      • occlusion of a coronary artery can be caused by
        • atheromatous plaque rupture with subsequent thrombi expansion
        • vasospasm
  • ECG Changes and STEMI
      • ECG Changes and STEMI
      • Infarction Location
      • Involved ECG Leads
      • Involved Coronary Artery
      • Inferior wall
      • II, III, and aVF
      • RCA
      • Antero-apical
      • V3 and V4
      • LAD (distal)
      • Antero-septal
      • V1 and V2
      • LAD
      • Antero-lateral
      • V5 and V6
      • LAD or LCX
      • Lateral
      • I and aVL
      • LCX
      • Posterior
      • ST depression and tall R waves in V1-3
      • V7-V9
      • Posterior descending artery
  • Evolution of MI
      • Morphological Myocardial Changes in an MI
      • Time
      • Gross Features
      • Light Microscopy
      • Complications
      • 0-24 hours
      • Initially no gross findings; however, over the course of the first 24 hours, dark mottling ensues
      • Early coagulation necrosis
      • Wavy fibers
      • Elongated myocytes
      • Neutrophil infiltration
      • Arrhythmia
      • Heart failure
      • 1-3 days
      • Mottling with a yellowish infarct center
      • Extensive coagulation necrosis
      • Brisk neutrophil infiltration
      • Fibrinous pericarditis
      • 3-14 days
      • 3-7 days
        • hyperemic with central yellowing
      • 7-10 days
        • yellow-tan with reddish tan margins
      • 10-14 days
        • reddish gray infarct borders
      • Macrophage infiltration and tissue granulation
      • Myocardial wall rupture
        • may lead to cardiac tamponade
      • Papillary muscle rupture
        • mitral regurgitation
        • posterolateral muscle rupture is more likely than anteromedial muscle rupture
      • Pseudoaneurysm of a ventricular wall
        • may rupture
      • 2 weeks - several months
      • 2-8 weeks
        • gray-white scar
      • > 2 months
        • complete scar
      • Collagenous scar
      • Dressler syndrome
      • Heart failure
      • True ventricular aneurysm
        • a thrombus may form
  • Imaging
    • Coronary angiography
      • indication
        • diagnostic study to assess coronary anatomy and to determine where the occlusion is
  • Differential
    • Unstable angina
      • differentiating factor
        • no elevation in cardiac biomarkers
    • Costochondritis
      • differentiating factor
        • chest pain that is reproducible with palpation
  • Complications
    • Heart failure
    • Sudden cardiac death
    • Arrhythmia
    • Myocardial stunning
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Question
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Cardiovascular | Myocardial Infarction
  • Cardiovascular
  • - Myocardial Infarction
23:44 min
5/16/2021
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